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Mother Margaret
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   Missing Key Value : en-US, 641 9/8/2006 12:00 PM (GMT -4)    Quote This PostAlert An Admin About This Post.
I'm wondering whether or not a premature baby who develops jaundice is at higher risk for autoimmune metabolic issues (autism, leukodystrophies, diabetes) than children who are not premature? .... and who do NOT develop jaundice?
 
I suspect exposures to 2-butoxyethanol in the family line and even exposures that can be in the air in one's home ... or that mom gets exposed to while carrying baby ... would be a primary cause of both.
 
My grandson who is not yet a month old was 3 weeks premature and developed jaundice ... "Oh, very commonplace" were the assuring words of the doctor.
 
Well, premature births and autism are also very commonplace
1 in 5 births are early; 1 in 166 births are children who become autistic (whereas it used to be 1 in 10,0000 not that long ago)
The statistics on many autoimmune issues are staggering
And birth defects are getting more "rare".
 
I suspect 2-butoxyethanol exposures can cause them all.
IF the pattern is there or in the family line.
 
This chemical should be suspect for organs shutting down, regardless of a person's age.  So, all the way from SIDS to the 3 soldiers who died last year after "flu like" symptoms
 
Body Temperature ... Diabetes ... Hypothalamus?

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Why I learned about 2-butoxyethanol - Blood in urine & small-sized RBCs are clues to its harm

Post Edited (Mother Margaret) : 9/8/2006 1:49:46 PM (GMT-4)

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